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Guidelines for the Diagnosis and Treatment of Tick-Borne lllness

Guidelines for the Diagnosis and Treatment of Tick-Borne Illness
UK HealthCare

Guidelines for the Diagnosis and Treatment of Tick-Borne Illness

Antimicrobial Stewardship Subcommittee · Pharmacy and Therapeutics Committee · University of Kentucky Health Care

AuthorsKatie Olney, PharmD, BCIDP; Thein Myint, MBBS; Sarah Jeter, PharmD, BCIDP, AAHIVP; Laura Stadler, MD; Joel Howard, MD; Morgan McCoy, MD
Target PopulationAdult patients undergoing workup for tick-borne illness
OverviewEvidence-based guidelines for the diagnosis and treatment of tick-borne illnesses.
Effective Date09/2020
Revised10/2022; 10/2024
Expiration10/2027 · Reviewed every 3 years
Primary OutcomeSelection of antimicrobial therapy and duration of therapy.

Purpose & Goals

To provide evidence-based information for the treatment of tick-borne infections in pediatric and adult patients admitted to the University of Kentucky Health Care System. The goals are to expedite collection of appropriate diagnostic studies, assist with early and appropriate diagnosis, and facilitate early initiation of appropriate antibiotics.

Background

Annual confirmed cases of Ehrlichiosis and Rocky Mountain Spotted Fever (RMSF) increased approximately eight-fold from 2000 to 2018 within the United States. Diagnosis of tick-borne illnesses is challenging because microbiologic testing is often not performed and clinical improvement frequently occurs before laboratory diagnosis. Ticks can harbor multiple pathogens, leading to simultaneous infections.

Kentucky has a significantly higher incidence of RMSF and ehrlichiosis than other common tick-borne illnesses (Babesia, Borrelia, Anaplasma, etc.). Both RMSF and ehrlichiosis are well known to cause severe morbidity including meningoencephalitis, cardiovascular failure, hepatic injury, acute respiratory distress syndrome, and renal failure.

Diagnosis & Initial Workup

Tick-borne illnesses can be difficult to diagnose and often present with non-specific symptoms. The classic triad is fever, headache, and rash. The most common presentation is an initial prodrome of flu-like illness including fever, myalgia, headache, malaise, nausea, vomiting, and diarrhea. Although most tick-borne illnesses occur between April and October, transmission can occur year-round in warmer climates.

Obtain a complete exposure history including: (1) history of tick bite, (2) exposure to environments with ticks, (3) identification of clusters of illness among contacts, and (4) travel history to endemic areas. Although tick exposure history is important, lack of a tick bite cannot serve as a diagnostic rule-out.

The decision to initiate treatment is commonly based on a clinical presumptive diagnosis from clinical findings and epidemiological risk factors. Treatment should not be withheld pending diagnostic test results — therapy should be initiated promptly and should not be discontinued until appropriate diagnostics return negative.

Infectious Diseases (ID) Consultation

An ID consultation is recommended in the following patients and/or clinical scenarios:

  • Pregnant patients
  • Pediatric patients
  • Immunocompromised patients
  • Patients with a life-threatening allergy to doxycycline
  • Lack of defervescence or improvement after >48 hours on doxycycline
  • Suspicion for Babesiosis
  • Suspicion for disseminated (neurological, cardiac, arthritic) Lyme disease
  • Equivocal diagnostic testing

Pediatric Concerns

Although tetracycline has historically been associated with teeth discoloration, doxycycline has not demonstrated the same affinity for calcium binding. Recent comparative data suggest doxycycline is not likely to cause visible teeth staining or enamel hypoplasia in young children (<8 years). Per the American Academy of Pediatrics (2018 Red Book), doxycycline may be administered for durations ≤21 days without regard to the patient's age. Doxycycline is the drug of choice for human ehrlichiosis and anaplasmosis and should be used regardless of patient age.

Figure 1: Acute Tick-Borne Illness Testing & Treatment Algorithm

Suspected tick-borne illness* Obtain complete exposure history: 1. History of tick bite 2. Exposure to environment with ticks 3. Cluster of Illness among contacts 4. Travel history to endemic areas. If available, send tick in sterile container to microbiology laba,b No Travel to Northeastern United States within 1-2 months from onset of symptoms Travel to Northeastern United States within 1-2 months from onset of symptoms Start Empiric Treatment with IV/PO Doxycyclined,e 100 mg q12h If no improvement or defeverscence on doxycycline >48 hours, consider alternative diagnosis CONSULT ID NO Presence of Erythema Migrans (bullseye rash)? YES • Obtain Anaplasma and Ehrlichia PCR • Obtain peripheral blood smear for Anaplasma and Ehrlichia • Obtain RMSF antibodyc • Consider Infectious Disease consultation Treat with IV/PO Doxycyclined,e 100 mg q12h for 10-14 days • Presumptive diagnosis of Lyme • No laboratory testing for Lyme required • Monitor for symptoms of other tick-borne illnesses NEGATIVE Anaplasma PCR Ehrlichia PCR • RMSF antibody titers POSITIVE (any of the following): Anaplasma PCR Ehrlichia PCR • Elevated RMSF antibody titers A negative result does NOT rule out the diagnosis of Ehrlichiosis, RMSF, or anaplasmosis. Doxycyclined,e should be continued for full duration in critically ill patients, those who improve on doxycycline, or if suspicion remains. May consider testing convalescent antibody serology 2 weeks from presentation if suspicion still present Continue treatment with IV/PO Doxycyclined,e 100mg q12h Refer to Table 3 for appropriate treatment duration *Suspicion of tick-borne illness should be based on the following: 1. Symptoms occurring during tick season (April-October): PLUS 2. Known tick exposure or environmental exposure in wooded areas Please see Table 1 for additional laboratory findings and symptoms suggestive of tick-borne illness a See Table 4 for SCM order set information; b Place in sterile, airtight container; c Consider obtaining Anaplasma antibodies if exposure occurred >1 week prior; d Pediatric dosing: Doxycycline 2.2mg/kg/dose IV/PO q12h (maximum dose: 100 mg); e Patients receiving doxycycline should be counseled to and avoid extended sun exposure due to risk of phototoxicity and take doses with food to decrease associated GI toxicity.

Table 1: Clinical Presentation of Selected Tick-Borne Illnesses

DiseaseVectorEndemic Region(s)Incubation Laboratory IndicesInitial PresentationCutaneous Features
Rocky Mountain Spotted Fever Wood tick (D. variabilis, D. andersoni) Five states (NC, OK, AR, TN, MO) account for 60% of cases 3–12 days Normal or ↑WBC; ↑ANC; ↑transaminases; thrombocytopenia; hyponatremia Fever, chills, severe headache, myalgia, nausea, vomiting, diarrhea, anorexia Maculopapular rash ~2–4 days after fever onset in most; may become petechial and involve palms and soles
Human Monocytic Ehrlichiosis Lone star tick (A. americanum); Blacklegged tick (I. scapularis) Southeast, Northeast, East of the Rocky Mountains, Pacific Coast 5–14 days ↓WBC; ↑transaminases; hyponatremia; thrombocytopenia Fever, headache, myalgia, nausea, diarrhea, vomiting, confusion, rash Variable pattern ~5 days after onset; may involve palms and soles. Rash in ~30% of adults, ~60% of children
Anaplasmosis Blacklegged tick (Ixodes scapularis) Upper Midwest, Northeast 5–14 days ↓WBC; ↑transaminases; thrombocytopenia Fever, headache, myalgia, nausea, diarrhea, vomiting Rare (<10%)
Babesiosis Blacklegged tick (Ixodes scapularis) Upper Midwest, Northeast ≥1–9 weeks ↑transaminases; ↓hematocrit; ↓reticulocytes; thrombocytopenia Chills, headache, myalgia, arthralgia None
Lyme Disease — Early Stage Blacklegged tick (Ixodes scapularis) Northeast, Mid-Atlantic, Upper Midwest 3–30 days None Fever, myalgia, headache, nausea, fatigue Erythema migrans
Lyme Disease — Early Disseminated Blacklegged tick (Ixodes scapularis) Northeast, Mid-Atlantic, Upper Midwest ≥2 weeks None Cardiac or musculoskeletal manifestations (see Table 2) None
Lyme Disease — Disseminated Blacklegged tick (Ixodes scapularis) Northeast, Mid-Atlantic, Upper Midwest ≥2 weeks None Neurologic or musculoskeletal manifestations (see Table 2) None

Source: CDC — cdc.gov/ticks/tickbornediseases

Lyme Disease

Per the CDC, Kentucky is a low Lyme incidence state with <10 confirmed cases/100,000 over the last three reporting years. Per the Kentucky Department of Public Health, there were only six confirmed Lyme cases in Kentucky in 2017, the majority in southern and western Kentucky.

  • Accurate history is crucial. Over-diagnosis of disseminated Lyme disease can lead to inappropriate use of health services and avoidable treatment-related illness.
  • For patients with neurological symptoms, the American Academy of Neurology and IDSA recommend parenteral regimens for the treatment of neuroborreliosis.
  • European studies suggest oral doxycycline and parenteral ceftriaxone may be equally effective for neuroborreliosis with facial palsy, though this remains untested in the US.

Given the complexities of diagnosing disseminated Lyme disease, ID consultation is advised.

Table 2: CDC Surveillance Definitions of Lyme Disease

Organ SystemDefinitionCaveats
Skin Erythema Migrans (EM): expanding skin lesion, often with partial central clearing (bullseye) around tick bite. Primary lesion must be ≥5 cm across largest diameter and diagnosis must be made by a physician. Annular erythematous lesions occurring within hours of tick bite represent hypersensitivity reactions and do NOT qualify as EM.
Musculoskeletal Recurrent, brief attacks (weeks or months) of objective joint swelling, sometimes followed by chronic arthritis. Chronic progressive arthritis not preceded by brief attacks, chronic symmetrical polyarthritis, or arthralgia/myalgia/fibromyalgia syndromes alone are NOT criteria.
Central Nervous System Any of the following alone or in combination: lymphocytic meningitis; cranial neuritis, particularly facial palsy (may be bilateral); radiculoneuropathy; encephalomyelitis. Headache, fatigue, paresthesia, or mildly stiff neck alone is NOT criteria. Encephalomyelitis must be confirmed by antibody production against Borrelia burgdorferi in CSF (higher titer in CSF than serum).
Cardiovascular Acute onset of high-grade (2nd or 3rd degree) atrioventricular (AV) conduction defect that resolves in days to weeks. Palpitations, bradycardia, bundle branch block, or myocarditis alone is NOT criteria.

Table 3: Selected Laboratory & Treatment Regimens

DiseaseDiagnostic Test(s)Confirmation & TimingTreatmentOther Considerations
Rocky Mountain Spotted Fever
Rickettsia rickettsii
Acute and convalescent serology 4-fold increase in antibody titer. IgM present day 3–8, peaks at 1 month; IgG present within 3 weeks, peaks 1–3 months. Doxycycline 100 mg IV/PO q12h. Duration: 7–14 days; treat at least 3 days after clinical improvement and fever resolution. Serology preferred. PCR may be positive in advanced disease. Cannot confirm with a single antibody test.
Human Monocytic Ehrlichiosis
Ehrlichia chaffeensis
E. chaffeensis PCR Chaffeensis DNA detected; or 4-fold titer increase; or morulae detected with positive antibody titer. Doxycycline 100 mg IV/PO q12h. Duration: 7–14 days; treat at least 3 days after improvement and fever resolution. CSF findings include pleocytosis and protein elevation. Negative PCR does not rule out disease.
Human Granulocytic Anaplasmosis
Anaplasma phagocytophilum
A. phagocytophilum PCR A. phagocytophilum DNA detected; or 4-fold titer increase; or morulae detected with positive antibody titer. Doxycycline 100 mg IV/PO q12h. Duration: 10–14 days. CSF usually normal. Negative PCR does not rule out disease. Cannot confirm with a single antibody test.
Babesiosis
Babesia microti
Peripheral blood smear of intraerythrocytic parasites (stat); Babesia PCR; antibody titer by IFA (supportive) Identification of Babesia in RBC; or Babesia DNA detected. Antibodies present within 4 weeks of onset of parasitemia. Atovaquone 750 mg q12h PLUS Azithromycin 1000 mg q24h. Duration: Consult ID. ID consult strongly recommended. Treat symptomatic patients with positive PCR or microscopic identification.
Lyme Disease
Borrelia burgdorferi
ELISA with Western Blot Acute/convalescent IgG serology in serum; or B. burgdorferi DNA in CSF or synovial fluid. Antibodies develop 2–4 weeks after bite. Erythema Migrans: Doxycycline 100 mg IV/PO q12h × 10 days.
Lyme Arthritis: × 28 days.
Lyme Carditis: × 14 days.
Neurological Lyme: × 14 days.
ID consult strongly recommended. EM at presentation = presumptive diagnosis. CSF/synovial testing not diagnostic without concurrent serum testing. Consider cardiology consult for cardiac involvement.

Note: Delay in treatment may result in severe illness or death. Antibodies may not be present in the first week of disease. Patients receiving doxycycline should avoid extended sun exposure (phototoxicity risk) and take doses with food to decrease GI toxicity.

Table 4: Diagnostic Order Identification in Epic

DiagnosticEpic Order
Identification of Tick Species"Arthropod Identification"
RMSF Acute & Convalescent Serology"Rickettsia (RMSF) Ab IgG and IgM w/ Reflex Titer"
Anaplasma phagocytophilum & Ehrlichia chaffeensis PCR"EHRLICHIA AND ANAPLASMA SPECIES BY REAL TIME PCR (SO)"
Serum Ehrlichia Antibodies"EHRL Chaffeensis AB"
Anaplasma & Ehrlichia Peripheral Blood Smear"Peripheral Blood Smear" → Click "Add Comments" → specify Anaplasma and Ehrlichia
Lyme ELISA with Western Blot"Borrelia burgdorferi VlsE1/pepC10 antibodies, total by ELISA with reflex to IgG and IgM (modified Two-Tier Testing) SO"
Lyme Disease CSF Antibodies"Borrelia species by PCR (Lyme Disease) (SO)"

Tick Bite Prevention & Prophylaxis

Prevention

Ticks live in grassy, wooded, or bushy areas — but many bites happen in people's own yards. Ticks can be carried indoors on pets, clothing, and gear. Clothing that doesn't need washing should be tumbled in a dryer on high for ten or more minutes to kill ticks; if washing is needed first, use hot water. After potential exposure, check the entire body and shower as soon as possible. Ticks prefer skin folds and hair-covered areas (in/around the ears, in the hair, between the thighs, under the armpit).

Prophylaxis

Before going outdoors, the CDC recommends treating clothing and gear with products containing 0.5% permethrin, and using insect repellents containing DEET, picaridin, IR3535, Oil of Lemon Eucalyptus (OLE), para-menthane-diol (PMD), or 2-undecanone. Avoid tall grasses and brushy areas, and stick to hiking trails. Talk to a veterinarian about prophylactic products for dogs; the CDC generally recommends against applying tick-prevention products to cats, which are extremely sensitive to many chemicals.

For prevention of Lyme disease after a recognized tick bite, routine antimicrobial prophylaxis or serologic testing is not recommended. In areas with high endemic rates of Lyme disease, a single 200 mg dose of doxycycline can be considered for high-risk bites where the tick has been attached for ≥36 hours.

References

  1. Wormser GP, et al. The Clinical Assessment, Treatment, and Prevention of Lyme Disease, Human Granulocytic Anaplasmosis, and Babesiosis: Clinical Practice Guidelines by the IDSA. Clin Infect Dis. 2006;43(9):1089–1134.
  2. CDC. Diagnosis and management of tickborne rickettsial diseases. MMWR. 2016;65(RR-2).
  3. Kimberlin DW, et al. Red Book: 2018–2021 Report of the Committee on Infectious Diseases. 31st ed. 2018.
  4. Bakken JS, et al. Human granulocytic anaplasmosis. Infect Dis Clin North Am. 2015:341–355.
  5. CDC. Updated CDC Recommendation for Serologic Diagnosis of Lyme Disease. MMWR. 2019;68(32):703.
  6. Ismail N, et al. Human Ehrlichiosis and Anaplasmosis. Clin Lab Med. 2010;30(1):261–292.
  7. CDC. Ehrlichiosis & RMSF Epidemiology and Statistics. 2020.
  8. Reid MC, et al. The consequences of overdiagnosis and overtreatment of Lyme disease. Ann Intern Med. 1998;128(5):354–362.
  9. Halperin JJ, et al. Practice Parameter: Treatment of nervous system Lyme disease. Neurology. 2007;69(1):91–102.
  10. Miller JM, et al. Guide to Utilization of the Microbiology Laboratory for Diagnosis of Infectious Diseases: 2018 Update. Clin Infect Dis. 2018;67(6):e1–e94.
  11. Kentucky Cabinet for Health and Family Services. Story-Map of Tickborne Disease in Kentucky.

Abbreviated reference list. See the original guideline document for the complete citations.

UK HealthCare · Antimicrobial Stewardship Subcommittee · Effective 09/2020, Revised 10/2024, Expires 10/2027
For more information: CDC Tick-Borne Diseases

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